We analyze the temporal behavior of the survival probability of an unstable$^6$Li Feshbach molecule close to the BCS-BEC crossover. We find differentinstances of nonexponential decay as the magnetic field approaches theresonance value, at which the molecule becomes stable. We observe a transitionfrom an exponential decay towards a regime dominated by a stretched-exponentiallaw.
Employing a new approach toward thermodynamic phase space, we investigate thephase transition, critical behavior and microscopic structure of higherdimensional black holes in an Anti-de Sitter (AdS) background and in thepresence of Power-Maxwell field. In contrast to the usual extended $P-V$ phasespace where the cosmological constant (pressure) is treated as a thermodynamicvariable, we fix the cosmological constant and treat the charge of the blackhole (or more precisely $Q^s$) as a thermodynamic variable. Based on this newstandpoint, we develop the resemblance between higher dimensional nonlinearblack hole and Van der Waals liquid-gas system. We write down the equation ofstate as $% Q^s=Q^s(T,\psi)$, where $\psi$ is the conjugate of $Q^s$, andconstruct a Smarr relation based on this new phase space as $M=M(S,P,Q^s)$,while $% s=2p/(2p-1)$ and $p$ is the power parameter of the Power-MaxwellLagrangian. We obtain the Gibbs free energy of the system and find aswallowtail behaviour in Gibbs diagrams, which is a characteristic offirst-order phase transition and express the analogy between our system and vander Waals fluid-gas system. Moreover, we calculate the critical exponents andshow that they are independent of the model parameters and are the same asthose of Van der Waals system which is predicted by the mean field theory.Finally , we successfully explain the microscopic behavior of the black hole byusing thermodynamic geometry. We observe a gap in the scalar curvature $R$occurs between small and large black hole. The maximum amount of the gapincreases as the number of dimensions increases. We finally find that characterof the interaction among the internal constituents of the black holethermodynamic system is intrinsically a strong repulsive interaction.
By extending the Cho decomposition method to G(2) gauge group, monopoles ofthis group are studied. Since SU(2) and SU(3) are subgroups of G(2),discussions are done mostly based on these subgroups of G(2). A direct relationbetween root vectors of G(2) and the associated magnetic charges is presentedby group theoretical issues. In addition, G(2) monopoles are obtained by anAbelian gauge fixing method, and it is shown that the results agree with theones we obtain by the Cho decomposition method.
Purpose During the ART era, persistent immune activation remains a significant challenge in people living with HIV (PLWH). Microbial translocation play an essential role in this setting. Probiotics have several immunological benefits which can reverse this process. The purpose of this paper is to investigate the safety and efficacy of probiotics on CD4 counts among Iranian PLWH. Design/methodology/approach In total, 50 PLWH with CD4 counts above 350 cells/mm 3 did not receive ART participated in a randomized, double-blind trial and underwent 24 weeks of treatment with either LactoCare® or placebo twice daily. CD4 counts of the patients were measured at baseline, 12 weeks and 24 later in the two groups. Side effects were measured monthly using a specific checklist. Findings The mean CD4 count of the patients showed a significant difference between the two groups after six months. Through six months follow up, the mean CD4 count of the patients showed a significant reduction as compared to the baseline in the placebo group; however, it did not show a significant difference in the probiotic group. Repeated Measures Anova test showed a significant effect for time × treatment interaction on the CD4 count during the trial course. No significant difference between the two groups concerning adverse events was reported. Originality/value It seems the use of probiotics in PLWH with a CD4 count above 350 cells/mm 3 who are not receiving antiretroviral drugs is safe and can reduce the devastating process of CD4+ T cells in these patients.